Obesity is a more imminent threat to one's health than the supposed long term effects of eating higher fat. A diet that is low enough in overall calories to cause weight loss is low enough in fat that it probably doesn't matter anyway. The diet after weight loss is another story and depends on goals and activity levels or other health considerations. ANYWAY, I'M NOT TALKING ABOUT WHAT I EAT, SO I'LL STOP HERE.
Hai. Kavik
There is alot of confusion surrounding eatings Fats, and Being-Fat..
Eating on the Input side of the equation will make a person LARGE in general..
This could be achieved with either Sugar or Fats.
However, even in people who can KEEP weight, their cholesterol will be significantly higher than a person eating less fat.
For example, triathlon athlete keeled over from a heart attack. On the outside, he's as fit as can be, However, when they autopsied the guy, His arteries were 90% clogged.
The contribution of fats consumption to atherosclerosis is independent of Exterior Large Appearance.
The way it works, is eating fat, elevates blood lipid levels. The lipids make it into the Endothelial layer. This layer is the root of artery disease pathology. The layer normally functions to dialate and contract the arteries, and produce lubricant gas nitric-oxide for blood flow.
However, with the lipid migrates into this layer, it becomes oxidized , damaged and scars over. The scaring is what produces the large blockages of diseased arteries..
With respect to Diabetes:
Elevated blood lipids deposit into muscle cells and the liver.
The muscle being one of the primary consumers of Sugars, NORMALLY, upon secretion of insulin (from your pancreas) will open its sugar channel, let the sugar in from the blood, and store it as glycogen, OR burn it for fuel.
The lipid deposit in muscle cells (intramyocellular lipid), hinders this process, making the muscles (of the entire body) insulin resistant.
And so the sugar Stays in the blood, and get to dangerous levels.
In the Liver, it works the same way, Normally The liver upon receiving insulin secreted by the pancreas will STOP releasing sugar. It is the organ that releases stored sugar from fats for daily operation when you haven't eaten.
But now that the fats (intramyocellular lipid) gums up the liver cells, they too do not respond to insulin, so the liver KEEPS injecting sugars..
This dual process of muscle insulin resistance and Liver insulin resistance is what creates the Runaway blood sugar levels responsible for Diabetes..
Again, this can also happen, independent of a Person actually Appearing Physically LARGE..